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Video Transcript: Can the Ketogenic Diet Starve Cancer Cells?
Charlene Bollinger: In this video excerpt with Toni Bark, learn more about the ketogenic diet and how you can use being in ketosis to starve cancer cells.
Ty Bollinger: Make sure you watch all the way down to the end to discover how ketones can help you keep healthy inflammation levels.
Dr. Toni Bark: I’m going to talk about reversing disease processes and using ketosis. And I want to mention something, because a lot of people tell me that they are on a ketogenic diet, and a lot of people have different ideas of what that means. Ketosis is something that happens in you. It’s not the diet.
There are diets that promote the ketogenic process and ketosis, but the reference is “are you fat burning?” That’s really what we’re talking about. Are you fat burning? Have you lowered your glucose levels? Are you keeping your insulin levels low?
So, a lot of people come up to me and tell me, “Oh, you know, I’m eating a ketogenic diet and I can’t lose weight and my sugar levels are high. I have no ketones” on however they are measuring it. And we will talk about that. Then I find out that they are eating a lot of animal protein. You know, a ketogenic diet is not a high protein diet. It’s a very moderate protein diet. It’s very muscle sparing.
So, there is a big misconception. People think that as long as they don’t eat carbs, they are eating a ketogenic diet. That’s not true. I just want to clear that from the beginning because that is a common question.
I want to go back to the metabolic theory of cancer, and Dr. Seyfried will be talking about this in more depth, but Otto Warburg, who I think coined this in the 30s, he studied cancer cells and he noticed that they swallowed up huge amounts of glucose and produced lactic acid, even in the presence of oxygen and utilized oxygen.
They also could use it without the oxygen, but the bottom line is that cancer cells require about 19 times the amount of glucose for the same amount of ATP production. And ATP, which is a term that has been thrown out at you, is the way we make fuel in the cells.
Our cells have mitochondria and I’m going to talk about mitochondria a lot today. Our cells have mitochondria and they are the fuel cell of your cells. That’s the engine. Think about it, some people have hybrid cars, right? So, if you have a hybrid car, your car can run on electricity and it can run on gas. Well, your engines can run on glucose and they can run on fats. And the fats are in the form of fatty acids in muscles and in hearts and ketones in brain and heart.
So, depending on the form of fatty acids, your cells can utilize fats. Cancer cells can’t utilize fats. And cancer cells have such a high glucose demand, that shifting your fuel source to fats is a way to trick the cancer cells and put a lot of pressure on them so they start dying.
Okay, so there’s emerging evidence that we’ve always heard that cancer is genetic, that it’s all these genetic defects. So, the drug companies and the big institutions, make a lot of money on the theory that this is individual genetic differences for each cancer.
But the evidence is pretty clear now that really, genetic defects are a byproduct of poor respiration at the cellular level. There are numerous studies to point this out and show this, which I am not going to go into these studies right now. You’re going to have to take my word for it, but I’m sure Thomas will go into it.
But cancer cells arise because there are problems at the mitochondria. There’s problems with metabolism. And the way that one of my friends who is a cannabis researcher, and cannabis is related to this story and I’ll get into that, talks about it. He says that when there is too much fuel coming down a pipeline, the pipeline has to split open and start dividing.
Inflammatory Theory of Disease. So, we know that in order for cancers to spread and for cancer to be there, there has to be some inflammation. And one of the things that I love about being in ketosis, meaning my body is making ketones from my fatty acids now, whether I have ingested them or I have just been storing them, is that the presence of ketones alone, especially in the brain, but everywhere, is going to reduce inflammatory markers. Inflammation is reduced.
So, we know with kids with chronic microglial activation, which we see from vaccine injury, from Roundup, injury to pesticides, they get chronic activation of the inflammatory cells in the brain. So, those cells that also have other things that they need to be doing are just constantly in the inflammatory mode. When we put those children in ketosis, we see an improvement immediately because those cells can now do other things and help grow, make synapsis in language and learning and social abilities.
Here are some studies that I’m quoting, on different studies where people are talking about colon cancer and all different inflammatory processes that are at tumorigenesis, meaning the production of tumors, that these are all related to chronic inflammation.
So, with my patients I look to see C-reactive protein. I look at hemoglobin A1c. To me, that gives me a lot of information about my patients. C-reactive protein is an inflammatory marker that you can see respond pretty quickly to putting a patient in ketosis. It will come way down. My patients who are on ketogenic diets, their CRPs are all in the 0 levels. 0.02, 3 or 4. Very low levels.
In effects on mitochondria and inflammatory markers, we’ve got ketosis, which I’m going to talk about how to do that. But when you’re in a state of ketosis and you’re limiting your carbohydrates and you’re limiting calories to some degree, but more importantly, and the verdict’s not out completely, is really keeping your insulin levels low.
Charlene Bollinger: Wow, that was very informative. Thank you Toni Bark and thank you out there for listening.
Ty Bollinger: If you enjoyed what you just heard, please leave some comments below for us.
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